Recovery of natural testosterone post cycle:
A brief run down of the body’s mechanism of producing testosterone:
Hypothalmus:
Using current and recent hormone levels this part of the brain releases LHRH – Luteinising Releasing Hormone, and
GNRH – Gonadotrophin Releasing Hormone
In turn these then act on a part of the brain called the:
Pituitary:
Here LHRH stimulates the Pit. To release LH
GHRH stimulates the Pit. To release FSH.
LH and FSH then act in the..
The Hypothalmus and Pituitary are otherwise known as the HPTA
Testes:
LH stimulates the leydig cells in the Testes to produce Testosterone
FSH stimulates the testes to produce Sperm.
Inhibition
Inhibition acts on all 3 levels of production,
Hypothalmus sensing high androgens releases less LHRH and GNRH
Pituitary both from reduced LHRH and GNRH, and also excessive estrogen from steroid use and
also insufficient estrogen from using too much anti-aromatose (femera and arimidex) as well as purely non aromatisable steroids which drive estrogen down.
This has the effect of down regulating the sensitivity of the Pituitary to LHRH so it is a double sword of inhibition.
Testes:
Without sufficient LH produced by the pituitary the Testes shut down and atrophy and no natural testorone is the end result.
PREVENTING INHIBITION:
A)
Minimising the effect on the HPTA
You cannot prevent inhibition at the HPTA but you can limit the effect somewhat by keeping estrogen under control.
If using large amounts of aromtising steroids, test, dianabol etc, using moderate amounts of anti-aromatose to keep estrogen in the normal range is wise. How do you know this – by blood tests.
Usually 0.25mg of Arimidex ED per 500mg of aromatising steroid is sufficient here.
So for example, 750mg of testosterone/week and 30mg of Dianabol ED = approx 1000mg of aromatising steroid so a dose of 0.5mg ED of Arimidex would be used
B)
Minimising the effect on the Testes during the cycle
When the testes atrophy especially for long periods it means post cycle there is substantial lag in picking up hormone production. It is far better to prevent rather cure Testicle atrophy.
A drug called HCG is used to do so. This mimics the effects of LH on the Testes, meaning despite using steroids, the Testes continue to produce testosterone, and donot atrophy, meaning post cycle they are up and running for a much better and fuller recovery.
To do this a regime of 500iu of HCG used twice each week for the duration of the cycle, ceasing its use 10 days before starting Clomid.
This is so that the boost in Testosterone Hcg causes can subside allowing recovery of the HPTA whilst being used as long as possible to prevent atrophy.
C)
Clomid Use to Restore the HPTA
The HPTA – the brain is the starting point for testosterone production, and without getting this crucial part back online you will not recover as fully or quickly.
As well as Testosterone and other ‘male’ steroids, the HPTA also uses levels of estrogen to regulate Testosterone production…
Estrogen is produced in the male by the aromatisation of testosterone to estrogen through the aromatose enzyme.
The HPTA sensing high estrogen assumes levels of Testosterone must be too high and ceases or reduces LH production.
To our advantage when the body senses low estrogen it ups the production of LH in the brain.
To achieve this effect anti estrogens, clomid and nolvadex are used
These have the effect of blocking the reception of estrogen in the HPTA so the brain is tricked into thinking LOW estrogen therefore BOOST LH and consequently Testosterone
Using Clomid and or Nolvadex
The levels of steroids must have cleared to a maximum of 100mg left in the body in order for androgen suppression to lift and any effect from
Anti estrogens to take effect..
Therefore using the half lives of the various steroid esters and amount used over the course of the cycle and the following tool::
http://powerboard.rockarfett.com/roidcalc/
calculate when steroid levels have reached 100mg total, or around 15mg a day in the body.
At this point Clomid should be started at 300mg on day 1, then 50mg ED thereafter for 4-6 weeks.
Nolvadex can be substituted at a loaded dose of 120mg day 1, then 20mg ED for 4-6 weeks
As there is some debate as to which is better some people choose to use both, in which case the abover regimes should be ran concurrently.
D)
The use of fast acting steroid esters to come off
If using long acting esters, 2-3 weeks must pass until the steroids leave the system sufficiently to allow HPTA recovery.
During this time the user is still suppressed completely, but not in an anabolic state.
This period can be maximised by using short acting steroids the 2-3 weeks longer esters take to clear.
This means the user is still very anabolic for the duration of the cycle until very close to Clomid therapy.
The following steroids are excellent during this time:
Testosterone Propionate – around 2/3 of the usual weekly testosterone dose is wise, given the remaining longer esters. EG 100mg prop 3 times/week for a cycle containing 500mg Test Cyp
Anavar – a superb if costly steroid, does not aromatise so no further impact on the HPTA from estrogen
PUTTING IT ALL TOGETHER, AN EXAMPLE:
The cycle:
Week 1-8 Testosterone Enanthate @ 500mg per week
Week 9-10 Testosterone Propionate 100mg 3 times/week
Week 1- 10 – 500iu of HCG twice each week
Week 11.5 – using
http://powerboard.rockarfett.com/roidcalc/ to calculate
Steroid levels have fallen to a total of 100mg/15mg per day in the body…
Start Clomid and/or Nolvadex
Recovery Of Natural Testosterone Post Cycle (pct)
Hybrid Mode
